{"id":11174,"date":"2012-09-27T14:12:59","date_gmt":"2012-09-27T18:12:59","guid":{"rendered":"https:\/\/www.saratoga.com\/living-well\/2012\/09\/the-power-of-the-gut-connections.html"},"modified":"2017-11-29T09:44:51","modified_gmt":"2017-11-29T14:44:51","slug":"the-power-of-the-gut-connections","status":"publish","type":"post","link":"https:\/\/www.saratoga.com\/living-well\/2012\/09\/the-power-of-the-gut-connections\/","title":{"rendered":"The Power of the Gut-Connections"},"content":{"rendered":"
by Sarah A LoBisco, ND<\/p>\n
This week’s blog highlights the connection between our gut’s critter population This week we are going to look at the impact of our gut bugs on:<\/strong><\/p>\n 1. <\/strong>Our 2. <\/strong>Inflammation<\/strong><\/p>\n 3. <\/strong>Brain Read more about these little microbes below:<\/strong><\/p>\n Gut Micoflora relates to A recent study demonstrated that the type of bugs resident in your \u00b7<\/strong>Elevated blood sugar (glucose of 100 mg\/dl or \u00b7<\/strong>High triglycerides (150 mg\/dl or higher)<\/strong><\/p>\n \u00b7<\/strong>Low HDL (under 50 mg\/dl for women, 40 mg\/dl \u00b7<\/strong>Weight gain (increased fat around the middle-as \u00b7<\/strong>Elevated blood pressure (130\/85 mmHg or According to the study:<\/strong><\/p>\n Obesity has Sources: <\/p>\n
\nto our health. The microflora that resides in our intestines outnumbers our
\ncells for a reason! They are critical regulators on how our body runs. <\/strong><\/p>\n
\nrisk for metabolic syndrome<\/strong><\/p>\n
\nand Mood<\/strong><\/p>\n
\nMetabolic Functions<\/u><\/strong><\/p>\n
\nintestines determines the risk for metabolic syndrome. This is a syndrome
\nthat consists of:<\/strong><\/p>\n
\nhigher)<\/strong><\/p>\n
\nfor men)<\/strong><\/p>\n
\nmeasured by waist circumference)<\/strong><\/p>\n
\nhigher)<\/strong><\/strong><\/p>\n
\nbeen linked to the human gut microbiota; however, the contribution of gut
\nbacterial species to the obese phenotype remains controversial because of
\nconflicting results from studies in different populations. To explore the
\npossible dysbiosis of gut microbiota in obesity and its metabolic
\ncomplications, we studied men and women over a range of body mass indices from
\nthe Old Order Amish sect, a culturally homogeneous Caucasian population of
\nCentral European ancestry. We characterized the gut microbiota in 310 subjects
\nby deep pyrosequencing of bar-coded PCR amplicons from the V1-V3 region of the
\n16S rRNA gene. Three communities of interacting bacteria were identified in the
\ngut microbiota, analogous to previously identified gut enterotypes. Neither BMI
\nnor any metabolic syndrome trait was associated with a particular gut
\ncommunity. Network analysis identified twenty-two bacterial species and four
\nOTUs that were either positively or inversely correlated with metabolic
\nsyndrome traits, suggesting that certain members of the gut microbiota may play
\na role in these metabolic derangements.<\/p>\n